Spike S1 protein found in Brain capillary endothelial and glial cells in dead vaccinated patient. No nucleocapsid of the virus found. NECROTIC ENCEPHALITIS, MYOCARDITIS--But, this also implicates pure #LongCovid imo, here's why

This immunohistochemistry slide shows spike S1 as brown granules in the capillary endothelial and glial cells in the Brain

This slide shows S1 causes endothelial SWELLING (tissue damage) and inflammatory response molecules to form in a cross-section of a capillary endothelial cells and glial cells in the Brain

This slide shows the same phenomemon in the heart.

The study states the spike protein was only found in areas with "acute inflammatory reactions" in endothelial, microglia, and astrocytes. Naturally, this is strong evidence of causation.

It also states that endothelial dysfunction is "highly involved in organ dysfunction during viral infections as it induces a pro-coagulant state, microvascular leak and organ ischemia."

This is one of the primary theories of #LongCovid as stated by the FLCCC and Bruce Patterson. Persistence of spike protein in monocytes carries the S1 throughout the body and across the blood brain barrier and also includes microglia activation. covid19criticalcare.com/treatment-prot…

However, besides LDN proposed for monocyte repolarization, there does not appear to be a therapy suggested for this theory.

Patterson's treatment theory suggests stopping attachment of monocytes to the endothelial cells through interruption of the fractalkine pathway based on my understanding. researchsquare.com/article/rs-134…

However, anticoagulation therapies could also theoretically inhibit spike binding to the endothelial cells and combined with potential degradation of S1 with fibrinolytic enzymes and autophagy, rid the body of S1. This has yet to be proven.

This study was found at MedCram, a youtube medical site. Please watch if you are interested. youtube.com/watch?v=i9JLZu…